Bromocriptine to combat trenbolone related side effects.
Bromo(2.5mgs/day) WILL take care of 99% of all Fina gyno sufferers.
Thats my assessment.
Oh, and drop the Vitex in the garbage. WORTHLESS.
Fina is a VERY POWERFUL anti-glucocorticoid, so what
exactly does it do to reduce endogeneous cortisone
levels?
There is only ONE mechanism:
A reduction in the TOTAL Free T4 and T3 levels within the
body.
T3 is HIGHLY catabolic to muscle, therefore by reducing it by(
take 45% as shown by Nandi as an example), you are
exerting a ridiculously high protein-sparing effect.
YES, thats right, Fina is not THAT anabolic IN VIVO, it is
far, and I do mean FAR more of an ANTI-CATABOLIC
AAS than anything else.
Ok, now lets back-track to the problem at hand.
TSH has been reduced by the trenbolone, which in
turns signals the thyroid to reduce endogeneously
produced levels of T3 and T4.
This reduction(As Nandi mentioned) causes a VERY
sharp drop in free T3 levels because of the reduction
in both the endogeneously produced T4 and T3.
(Remember that 80% of the free T3 is produced from
the metabolically inactive T4)
These dimished levels of T3,T4 cause Thyrotropin Releasing
Hormone(TRH) to become OVER-STIMULATED.
In essence, this is your bodies feed-back loop to reduced
thyroid hormones, due to a GLUCO-CORTICOID suppresive
effect. This is however NOT like hypothyroidic patients
who have a naturally defective(or damaged) thyroid.
When TRH becomes over-stimulated the net effect is
a VERY sharp increase in prolactin levels.
Critical here.....
I.E. YOU BEGIN TO LACTATE!!!!!
Now, herein lies the problem. Everybody is bio-chemically
different, therefore the TRH increase is EXTREMELY
broad-spectrum.
While someone will stimulate TRH say X% and ultimately
cause a rise in prolactin of say Y% with a daily
dosage of 50mg ED of Fina, another person will
cause a 2X% rise in TRH and 2Y+% rise in prolactin
which will invariably lead to gyno.
This is just genetics. Nothing can be done about this.
However, there are ways to combat prolactin-elevations:
This btw, HAS TO BE EXACT. If you over-dose you cause
a progestenic shift due to severely inhibited prolactin levels,
or if you under-dose you run the risk of getting prolactin
induced gyno.
As a note: PROGESTERONE does NOT, I repeat NOT come into
play with Fina at all. It only becomes into play when you're
trying to inhibit prolactin synthetically.
The only thing that can combat Fina-induced Gyno is:
1. 2.5mgs Bromocriptine broken down to 1.25mgs 2X/day
AM and PM.
Bromo(2.5mgs/day) WILL take care of 99% of all Fina gyno sufferers.
Thats my assessment.
Oh, and drop the Vitex in the garbage. WORTHLESS.
Fina is a VERY POWERFUL anti-glucocorticoid, so what
exactly does it do to reduce endogeneous cortisone
levels?
There is only ONE mechanism:
A reduction in the TOTAL Free T4 and T3 levels within the
body.
T3 is HIGHLY catabolic to muscle, therefore by reducing it by(
take 45% as shown by Nandi as an example), you are
exerting a ridiculously high protein-sparing effect.
YES, thats right, Fina is not THAT anabolic IN VIVO, it is
far, and I do mean FAR more of an ANTI-CATABOLIC
AAS than anything else.
Ok, now lets back-track to the problem at hand.
TSH has been reduced by the trenbolone, which in
turns signals the thyroid to reduce endogeneously
produced levels of T3 and T4.
This reduction(As Nandi mentioned) causes a VERY
sharp drop in free T3 levels because of the reduction
in both the endogeneously produced T4 and T3.
(Remember that 80% of the free T3 is produced from
the metabolically inactive T4)
These dimished levels of T3,T4 cause Thyrotropin Releasing
Hormone(TRH) to become OVER-STIMULATED.
In essence, this is your bodies feed-back loop to reduced
thyroid hormones, due to a GLUCO-CORTICOID suppresive
effect. This is however NOT like hypothyroidic patients
who have a naturally defective(or damaged) thyroid.
When TRH becomes over-stimulated the net effect is
a VERY sharp increase in prolactin levels.
Critical here.....
I.E. YOU BEGIN TO LACTATE!!!!!
Now, herein lies the problem. Everybody is bio-chemically
different, therefore the TRH increase is EXTREMELY
broad-spectrum.
While someone will stimulate TRH say X% and ultimately
cause a rise in prolactin of say Y% with a daily
dosage of 50mg ED of Fina, another person will
cause a 2X% rise in TRH and 2Y+% rise in prolactin
which will invariably lead to gyno.
This is just genetics. Nothing can be done about this.
However, there are ways to combat prolactin-elevations:
This btw, HAS TO BE EXACT. If you over-dose you cause
a progestenic shift due to severely inhibited prolactin levels,
or if you under-dose you run the risk of getting prolactin
induced gyno.
As a note: PROGESTERONE does NOT, I repeat NOT come into
play with Fina at all. It only becomes into play when you're
trying to inhibit prolactin synthetically.
The only thing that can combat Fina-induced Gyno is:
1. 2.5mgs Bromocriptine broken down to 1.25mgs 2X/day
AM and PM.
