S
saudades
Guest
I have been researching myostatin inhibitors for some time now. The thought occurred to me that if say for an example, follistatin (which has been shown to inhibit myostatin) became mainstream and people were using it, assuming that one gains a lot of muscle from using it--one wouldn't even need to use AAS to get big. (I'm sure there'd still be people who use, but it wouldn't necessarily be needed--that's not the scope of this post anyway.) Follistatin does not have any side effects (that we know of) so if people were becoming more muscular from it, I'm curious how that would affect people's perceptions of AAS. What do you think?